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Patients may present with weakness progressing to flaccid paralysis, paraesthesia, or depressed deep tendon reflexes.Alternatively, the clinical picture can be overshadowed by the primary illness causing hyperkalaemia.It is seen more commonly as a consequence of asphyxia, which accounts for most of the non-cardiac causes of cardiac arrest.

The better outcomes for OHCA victims receiving compression-only CPR Survival after cardiac arrest from asphyxia is rare and most survivors sustain severe neurological injury.Of five published series that included a total of 286 patients with cardiac arrest following hanging where CPR was attempted (this was attempted in only about 16% of cases), there were just six (2%) survivors with a full recovery; 11 other survivors all had severe permanent brain injury.The precise values that trigger treatment decisions will depend on the patient's clinical condition and rate of change of electrolyte values.There is little or no evidence for the treatment of electrolyte abnormalities during cardiac arrest.This is the most common electrolyte disorder associated with cardiac arrest.

It is usually caused by impaired excretion by the kidneys, drugs or increased potassium release from cells and metabolic acidosis.

Guidance during cardiac arrest is based on the strategies used in the non-arrest patient.

When possible, identify and treat life-threatening electrolyte abnormalities before cardiac arrest occurs.

The third part is focused on patients with specific conditions, and those with certain long-term comorbidities where a modified approach and different treatment decisions may be necessary.

Cardiac arrest caused by pure hypoxaemia is uncommon.

Hyperkalaemia occurs in up to 10% of hospitalised patients.; in practice, hyperkalaemia is a continuum.